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	<title>Institute of Paediatric Orthopaedic Disorders (IPOD) &#187; Patient&#8217;s Section</title>
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		<title>Ponseti&#8217;s treatment for Clubfoot</title>
		<link>http://ipodindia.org/2010/07/ponsetis-treatment-for-clubfoot/</link>
		<comments>http://ipodindia.org/2010/07/ponsetis-treatment-for-clubfoot/#comments</comments>
		<pubDate>Fri, 16 Jul 2010 14:04:30 +0000</pubDate>
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				<category><![CDATA[Club foot]]></category>
		<category><![CDATA[IPOD Focus]]></category>
		<category><![CDATA[Patient's Section]]></category>

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		<description><![CDATA[Click here to download Ponseti clubfoot manual 

]]></description>
			<content:encoded><![CDATA[<p><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/ctev1.pdf" target="blank">Click here to download Ponseti clubfoot manual</a> </p>
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		<title>Idiopathic Toe Walking</title>
		<link>http://ipodindia.org/2010/07/idiopathic-toe-walking/</link>
		<comments>http://ipodindia.org/2010/07/idiopathic-toe-walking/#comments</comments>
		<pubDate>Tue, 13 Jul 2010 01:47:42 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[IPOD Focus]]></category>
		<category><![CDATA[Patient's Section]]></category>

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		<description><![CDATA[1. What happens if we do not treat ITW

J Am Podiatr Med Assoc. 1997 Jan;87(1):17-22.
Effect of  persistent toe walking on ankle equinus. Analysis of 60 idiopathic toe  walkers.
Sobel E, Caselli MA, Velez Z.
New York College of  Podiatric Medicine, New York, USA.
Abstract
Sixty idiopathic  toe walkers (age range 1 to 15 years) were [...]]]></description>
			<content:encoded><![CDATA[<p><strong>1. What happens if we do not treat ITW<br />
</strong><br />
J Am Podiatr Med Assoc. 1997 Jan;87(1):17-22.<br />
Effect of  persistent toe walking on ankle equinus. Analysis of 60 idiopathic toe  walkers.</p>
<p>Sobel E, Caselli MA, Velez Z.</p>
<p>New York College of  Podiatric Medicine, New York, USA.<br />
Abstract</p>
<p>Sixty idiopathic  toe walkers (age range 1 to 15 years) were evaluated to determine the  natural history of toe-to-toe gait and the relationship between the  range of ankle dorsiflexion and increasing age. The majority of toe  walkers had a normal birth weight (average 7.06 pounds), walked on time  (average 11.14 months), began toe walking immediately (87%), stood  plantigrade (90%), were able to demonstrate a heel-toe gait (88%), and  toe walked intermittently (68%). Forty-six percent of all toe walkers  were found to have 0 degree or less of passive ankle dorsiflexion. <span style="background-color: #ff9966;">Equinus toe walkers (mean  dorsiflexion -5.2 degrees) had significantly less dorsiflexion than the  remaining toe walkers</span> (mean dorsiflexion 16.9 degrees; p &lt;  0.01). An average of 12 degrees of dorsiflexion was resent in the 1-to  2-year age group, which gradually diminished to -4 degrees in the 6- to  15-year age group. <span style="background-color: #cc6600;">It  appears that there may be a relationship between persistent toe walking  and the development of ankle equinus in some children and therefore  interventions should be considered to inhibit the toe walking  progression.</span></p>
<p><em style="color: #6666cc;"><strong>The  article concludes that toe walkers where there is equinus deformity need  treatment to improve dorsiflexion<br />
</strong></em><br />
<strong>2. What are the results of conservative treatment and serial casting?<br />
</strong><br />
Acta Paediatr. 2004 Feb;93(2):196-9.<br />
The natural history of  idiopathic toe-walking: a long-term follow-up of fourteen conservatively  treated children.<br />
Hirsch G, Wagner B.</p>
<p>Department of  Paediatric Orthopaedics, Astrid Lindgren&#8217;s Children&#8217;s Hospital,  Karolinska sjukhuset, Stockholm, Sweden. georg.hirsch@ks.se<br />
Abstract</p>
<p>AIM:  To determine the long-term results after conservative treatment  (physiotherapy, casting, orthoses, or a combination of these) of  idiopathic toe-walking (ITW). METHODS: Tiptoe-walking is diagnosed as  idiopathic (habitual) if no signs of neurological, orthopaedic, or  psychiatric disease are detected. The diagnosis is one of exclusion.  Sixteen former patients with ITW, all now at least 13 y old, were asked  to participate in a follow-up investigation 7-21 y after being first  diagnosed. Two cases were excluded because heel-cord lengthening had  been performed later on in other hospitals. The remaining 14 patients  completed a questionnaire. Eleven patients consented to a clinical  examination, during which they were videotaped and their active and  passive ankle-joint dorsiflexion measured. These data were compared with  the assessment at the initial evaluation. In one instance, the  toe-walking ceased after conservative treatment (plaster cast). In all  other cases the toe-walking pattern recurred. RESULTS: At follow-up  three patients showed some toe-walking when they were unobtrusively  observed. When videotaped, they did not toe-walk, although a distinct  heel-strike was missing. The remaining eight patients all walked with a  heel-strike. Two patients had slight symptoms possibly related to  toe-walking. No fixed contracture was present at the first evaluation,  and none was found at follow-up.<span style="color: #33ccff;"> <span style="background-color: #ffff99; color: #000000;">There was no systematic change in  ankle-joint dorsiflexion from initial assessment to follow-up  examination</span></span>. CONCLUSION: <span style="background-color: #ff9900;">Non-surgical treatment of ITW does not have a  lasting effect and the long-term results in this study are considered to  reflect the natural history, i.e. the toe-walking pattern eventually  resolves spontaneously in the majority of children. Surgical treatment  of ITW should be reserved for the few cases with a fixed ankle-joint  contracture.</span></p>
<p><em style="color: #6666cc;"><strong>The article concludes that toe  walkers where there is equinus deformity need treatment to improve  dorsiflexion<br />
</strong></em><br />
Acta Orthop Belg.  2006 Dec;72(6):722-30.<br />
Serial casting in the  treatment of idiopathic toe-walkers and review of the literature.</p>
<p>Fox  A, Deakin S, Pettigrew G, Paton R.</p>
<p>Blackburn Royal Infirmary,  Blackburn, United Kingdom. annafox2k@btopenworld.com<br />
Abstract</p>
<p>Idiopathic  toe-walking is defined as persistent toe-walking in a normal child in  the absence of developmental, neurological or neuromuscular conditions.  True idiopathic toe-walking is a rare referral, representing  approximately 1:100 new patients seen in the Paediatric Orthopaedic  Clinic. A prospective study of idiopathic toe-walking (ITW) was  organised between 1999 and 2003. Patients underwent full history,  neurological examination and assessment of ankle dorsiflexion, followed  by below-knee weight-bearing casting. Forty four developmentally normal  children with no delay in walking age were in this study. There was an  age range on presentation from 2 years to 14 years 4 months, with median  60.5 months. Sixty eight percent were male. Thirty four percent had a  family history of the condition. <span style="background-color: #ffcc99;">Following casting, 66% of patients had  improved gait on patient and clinician determined outcomes, with the  majority of children ceasing to toe-walk. Ankle dorsiflexion  significantly improved in those children who were successfully treated  (p = 0.001).</span></p>
<p><strong><em><span style="color: #3366ff;">The article conclused that  in one third of patients the serial casting did not work these are  likely to be patients with ankle equinus</span><br style="color: #3366ff;" /> </em></strong><br />
<strong>3. What is the outcome of surgery for ITW<br />
</strong><br />
J Pediatr Orthop.  2006 May-Jun;26(3):336-40.<br />
Outcome of  patients after Achilles tendon lengthening for treatment of idiopathic  toe walking.</p>
<p>Hemo Y, Macdessi SJ, Pierce RA, Aiona MD, Sussman  MD.</p>
<p>Dana Children&#8217;s Hospital, Tel Aviv Medical Center, Tel Aviv,  Israel.<br />
Abstract</p>
<p>Fifteen children who were diagnosed with  idiopathic toe walking that cannot be corrected by nonoperative  treatment were assessed by clinical examination and computer-based gait  analysis preoperatively and approximately 1 year after Achilles tendon  lengthening. Passive dorsiflexion improved from a mean plantarflexion  contracture of 8 degrees to dorsiflexion of 12 degrees after surgery.  Ankle kinematics normalized, with mean ankle dorsiflexion in stance  improving from -8 to 12 degrees and maximum swing phase dorsiflexion  improving from -20 to 2 degrees. Peak ankle power generation increased  from 2.05 to 2.37 W/kg but did not reach values of population norms. No  patient demonstrated clinically relevant triceps surae weakness or a  calcaneal gait pattern. Seven patients had a stance phase knee  hyperextension preoperatively, and 6 of these corrected after surgery. <span style="background-color: #ff9900;">Achilles tendon lengthening  improves ankle kinematics without compromising triceps surae strength;  however, plantarflexion power does not reach normal levels at 1 year  after surgery.</span><br style="background-color: #ff9900;" /><br />
<strong style="color: #6633ff;"><em>The article conclused that  surgery in selected cases who do not improve with conservative treatment  improves ankle dorsiflexion and way the child walks inclusing the  energy efficiency. There may be calf weakness post operatively which  corrects in one year time</em></strong></p>
<p>J Pediatr Orthop.  2001 Nov-Dec;21(6):790-1.<br />
Simplified approach to  idiopathic toe-walking.</p>
<p>Kogan M, Smith J.</p>
<p>Primary  Children&#8217;s Medical Center, Salt Lake City, Utah, USA.<br />
Abstract</p>
<p>Controversy  exists as to the treatment of idiopathic toe-walking (ITW). Since 1993,  the authors have managed children with ITW using an outpatient  percutaneous lengthening of the Achilles tendon, followed by placement  of below-knee walking casts for 4 weeks. The authors reviewed 15  children who were treated for ITW with percutaneous Achilles tendon  lengthening between 1993 and 1999. Ten of the 15 patients could be  contacted for a follow-up survey. None of the parents stated that their  child&#8217;s toe-walking had recurred. There were no painful scars. All of  the children were able to keep up with other children and did not notice  any calf weakness. Two children had occasional Achilles tendinitis,  which was relieved with antiinflammatory medications. All parents were  satisfied with their child&#8217;s outcome. <span style="background-color: #ff9966;">The authors believe that percutaneous  tendo-Achilles lengthening in ITW greatly simplifies the management of  ITW.</span></p>
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		<title>Current management of cerebral palsy</title>
		<link>http://ipodindia.org/2010/06/current-management-of-cerebral-palsy/</link>
		<comments>http://ipodindia.org/2010/06/current-management-of-cerebral-palsy/#comments</comments>
		<pubDate>Fri, 18 Jun 2010 05:40:06 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Cerebral palsy]]></category>
		<category><![CDATA[IPOD Focus]]></category>
		<category><![CDATA[Patient's Section]]></category>

		<guid isPermaLink="false">http://ipodindia.org/?p=426</guid>
		<description><![CDATA[The article explores the curent management of cerebral palsy]]></description>
			<content:encoded><![CDATA[<div><strong>What causes deformity in cerebtra palsy?</strong></div>
<div> </div>
<div>a.The spastic muscles are not able to relax and cuase unequal muscle forces leading to <strong>dynamic deformities</strong>.</div>
<div>b.The muscles under tension can not grow as well as the normal muscles This causes relative shortening of muscles and <strong>fixed contractures (secondary deformity)</strong></div>
<div>c. As the child&#8217;s age advances there are secondary contractures in joints and rotational deformities in bones which cause <strong>lever arm dysfunction (tertiary deformities)</strong></div>
<div> </div>
<div><strong>What is lever arm dysfunction?</strong></div>
<div> </div>
<div>Disruption in the moment generation of a muscle joint complex because of an ineffective lever or moment arm despite normal muscle force. Four types of lever arm dysfunction are described by Gage: malrotation, loss of a stable fulcrum, loss of bony rigidity, and lever arm shortening. The result of lever arm dysfunction is functional weakness and decreased power production</div>
<div> </div>
<div><strong>How does one assess the extent of the deformities</strong></div>
<div> </div>
<div>a. The amount of spasticity and muscle shortening is assessed by tardeau scale</div>
<div>b. The amount of bony and joint deformities is assessed by clinical evaluation, xrays and CT scan</div>
<div> </div>
<div><strong>How do we at IPOD decide appropriate treatment for CP patients</strong></div>
<div><strong></strong> </div>
<div>a. Small children where spasticity is an issue and who do not have contractures or muscle shortening. The spasticity is controlled with therapy, splints and botulinum toxin injection</div>
<div><strong></strong> </div>
<div>b. Children in age group 4-6 years with spasticity and muscle shortening: We treat them with a combined approach spasticity is treated with botulinum toxin injection and the shortening is treated with PAS (percutaneous aponeurotic slide). PAS is a percutaneous minimally invasive surgery where muscle covering is incised with a special needle to elongate the muscles yet retaining the power. The lower limbs are immobilised in plaster cast for 2 weeks sfter the procedure and then exercises started. The advantage of the procedure is that there are no stitches or scars, the procedure takes a short time of anaesthesia, the muscle healing is faster, The muscle power is retained and the need for repeated injections is avoided.</div>
<div> </div>
<div>c. the children with lever arm disorders need single event multilevel surgery with percutaneous bony osteotomies to correct the angulation and rotation along with multilevel aponeurotic releases. the advantage of percutaneous bony osteotomies again is shorter healing time, smaller scars and early recovery.</div>
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		</item>
		<item>
		<title>Technique of Hemiepiphysiodesis with E plate</title>
		<link>http://ipodindia.org/2009/10/technique-of-hemiepiphysiodesis-with-e-plate/</link>
		<comments>http://ipodindia.org/2009/10/technique-of-hemiepiphysiodesis-with-e-plate/#comments</comments>
		<pubDate>Sat, 31 Oct 2009 15:24:51 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Patient's Section]]></category>

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		<description><![CDATA[Dr Taral V Nagda
Dr Rajeev Nirawane
Institute of Paediatric Orthopaedic Disorders
 www.ipodindia.org
taralnagda@gmail.com
 Indications

Correcting angular deformity in a growing child with open physis



Any age/ Size
Any accessible physis 
Any diagnosis including sick physis 
1 year of growth remaining
Any plane frontal sagital oblique

Contraindications

Closed Physis 
Skeletal Maturity
Physiological Deformities

Equipments needed

C ARM
Radiolucent table
1mm and 1.5 mm guide wires
E plate
4.5mm self tapping cannulated [...]]]></description>
			<content:encoded><![CDATA[<p><strong>Dr Taral V Nagda<br />
Dr Rajeev Nirawane<br />
<em>Institute of Paediatric Orthopaedic Disorders</em></strong><br />
<strong><em> </em><a href="http://www.ipodindia.org/">www.ipodindia.org</a><br />
<a href="mailto:taralnagda@gmail.com">taralnagda@gmail.com</a></strong></p>
<h2><span style="color: #800080;"> Indications</span></h2>
<ul>
<li><strong>Correcting angular deformity in a growing child with open physis<br />
</strong></li>
</ul>
<ul>
<li><strong>Any age/ Size</strong></li>
<li><strong>Any accessible physis </strong></li>
<li><strong>Any diagnosis including sick physis </strong></li>
<li><strong>1 year of growth remaining</strong></li>
<li><strong>Any plane frontal sagital oblique</strong></li>
</ul>
<h2><span style="color: #800080;">Contraindications</span></h2>
<ul>
<li><strong>Closed Physis </strong></li>
<li><strong>Skeletal Maturity</strong></li>
<li><strong>Physiological Deformities</strong></li>
</ul>
<h2><span style="color: #800080;">Equipments needed</span></h2>
<ul>
<li><strong>C ARM</strong></li>
<li><strong>Radiolucent table</strong></li>
<li><strong>1mm and 1.5 mm guide wires</strong></li>
<li><strong>E plate</strong></li>
<li><strong>4.5mm self tapping cannulated screws (15-30mm)</strong></li>
<li><strong>Stop Drill</strong></li>
</ul>
<h2><span style="color: #993366;">Surgical steps</span></h2>
<h2><span style="color: #0000ff;">Step 1 : LOCALIZATION OF PHYSIS</span></h2>
<ul>
<li><strong>Under image intensifier 1.5 mm guide wire is passed at the center of physis</strong></li>
<li><strong>Confirmed in both AP &amp; Lat </strong> <strong>views</strong></li>
</ul>
<p><strong> </strong></p>
<p><strong> <a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard013.jpg"><img class="alignnone size-medium wp-image-374" title="Clipboard01" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard013-300x224.jpg" alt="Clipboard01" width="299" height="225" /></a></strong></p>
<h2><span style="color: #0000ff;">Step 2: SKIN INCISION</span></h2>
<ul>
<li><strong>About 1.5 cm centered over guide wire</strong></li>
<li><strong>The incision is deepened till perichomdrium</strong></li>
<li><strong>E plate is positioned with its central hole sliding over the guide wire over the perichondrium</strong></li>
<li><strong>The plate should be along the midline of the bone axis on lateral view</strong></li>
</ul>
<h2><span style="color: #0000ff;">Step 3 GUIDE WIRES</span></h2>
<ul>
<li><strong>2 guide wires are passed  through the holes on either sides of the physis </strong></li>
<li><strong>They  need not be parallel but  care should be taken is not to violate the physis </strong></li>
</ul>
<p><strong> <a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard042.jpg"><img class="alignnone size-medium wp-image-377" title="Clipboard04" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard042-300x224.jpg" alt="Clipboard04" width="300" height="224" /></a></strong></p>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard022.jpg"><img class="alignnone size-medium wp-image-399" title="Clipboard02" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard022-300x224.jpg" alt="Clipboard02" width="300" height="224" /></a></h2>
<h2><strong><img class="alignnone size-medium wp-image-378" title="Clipboard05" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard051-300x224.jpg" alt="Clipboard05" width="300" height="224" /></strong></h2>
<h2><span style="color: #0000ff;">Step 4 SELF TAPPING CANNULATED SCREWS</span></h2>
<ul>
<li><strong>Screw length is measured</strong></li>
<li><strong>Should not be too short</strong></li>
<li><strong>Generally 15-30 mm screws are sufficient</strong></li>
<li><strong>Cortex is drilled</strong></li>
<li><strong>Self tapping screws are passed</strong></li>
</ul>
<p><strong> </strong></p>
<p><strong> <a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard081.jpg"><img class="alignnone size-medium wp-image-380" title="Clipboard08" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard081-300x224.jpg" alt="Clipboard08" width="305" height="222" /></a></strong></p>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard094.jpg"><img class="alignnone size-medium wp-image-398" title="Clipboard09" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard094-300x247.jpg" alt="Clipboard09" width="300" height="247" /></a></h2>
<h2><span style="color: #0000ff;">Step 5 Closure and Dressing</span></h2>
<p><strong>The closure is done in layers and compression dressing is applied.</strong></p>
<p><strong>No cast or immobilization is needed</strong></p>
<p><strong>Full weight bearing supported walking and knee mobilization are started the next day</strong></p>
<p><strong>Post op Protocol</strong></p>
<ul>
<li><strong>Day care surgery</strong></li>
<li><strong>No immobilisation </strong></li>
<li><strong>Immediate mobilisation </strong></li>
<li><strong>Xrays once in 3 months </strong></li>
<li><strong>Implant removal once the deformity is corrected</strong></li>
</ul>
<h2><span style="color: #0000ff;">Case report</span>-</h2>
<p><strong>8 yr old girl with renal osteodystrophy with genu valgum</strong></p>
<p><strong> <a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard10.jpg"><img class="alignnone size-medium wp-image-385" title="Clipboard10" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard10-300x290.jpg" alt="Clipboard10" width="321" height="293" /></a><img class="alignnone size-medium wp-image-384" title="Clipboard11" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard113-270x300.jpg" alt="Clipboard11" width="221" height="290" /></strong></p>
<p><strong> </strong></p>
<h2><span style="color: #0000ff;">Immediate post op xray</span></h2>
<p><strong> </strong></p>
<h2><span style="color: #0000ff;">3 month follow up</span></h2>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard12.jpg"><img class="alignnone size-medium wp-image-382" title="Clipboard12" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard12-200x300.jpg" alt="Clipboard12" width="203" height="300" /></a></h2>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard181.jpg"><img class="alignnone size-medium wp-image-404" title="Clipboard18" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/Clipboard181-216x300.jpg" alt="Clipboard18" width="214" height="298" /></a></h2>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<h2><span style="color: #0000ff;">6 month follow up</span></h2>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<p><strong> </strong></p>
<h2><span style="color: #0000ff;">Advantages of E plate over </span><span style="color: #0000ff;">staples</span></h2>
<ul>
<li>Flexibility</li>
<li>Unconstrained implant</li>
<li>Load sharing</li>
<li>One plate per physis</li>
<li>Fully threaded cannulated screws resist pull-out forces</li>
<li>Diverging screws function like a hinge to gently guide natural growth</li>
<li>Easy to remove</li>
</ul>
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		<title>Camp for physically challanged at Dharampur Gujrat</title>
		<link>http://ipodindia.org/2009/10/camp-for-physically-challanged-at-dharampur-gujrat/</link>
		<comments>http://ipodindia.org/2009/10/camp-for-physically-challanged-at-dharampur-gujrat/#comments</comments>
		<pubDate>Sat, 10 Oct 2009 19:20:19 +0000</pubDate>
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				<category><![CDATA[IPOD News]]></category>
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		<description><![CDATA[A camp for physically challanged children is organised by Shrimad Rajchandra Hospital Dharampur Near Valsad Gujarat on 5 and 6 Feb 2010. The children will be evaluated on the 14th and the surgeries will be performed on 15th October. For details contact Nivedita on 09321329888
]]></description>
			<content:encoded><![CDATA[<p>A camp for physically challanged children is organised by Shrimad Rajchandra Hospital Dharampur Near Valsad Gujarat on 5 and 6 Feb 2010. The children will be evaluated on the 14th and the surgeries will be performed on 15th October. For details contact Nivedita on 09321329888</p>
]]></content:encoded>
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		<title>Pulled Elbow in Children</title>
		<link>http://ipodindia.org/2009/10/pulled-elbow/</link>
		<comments>http://ipodindia.org/2009/10/pulled-elbow/#comments</comments>
		<pubDate>Wed, 07 Oct 2009 02:03:14 +0000</pubDate>
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				<category><![CDATA[IPOD Focus]]></category>
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		<description><![CDATA[Pulled Elbow or “Nursemaids Elbow” is common in young children between 1 and 4 years of age.It is rare beyond the age of 6 years. It is due to the annular ligament of the radial head becoming stretched and entrapped.The treatment is by reduction in flexion and supination]]></description>
			<content:encoded><![CDATA[<h2>What is Pulled Elbow?</h2>
<p><!-- <ecm_dcontent> &#8211;>  					 					  								<!-- <ecm_dcontent> &#8211;>Pulled Elbow or “Nursemaids Elbow” is common in young children between 1 and 4 years of age.It is rare beyond the age of 6 years. It is due to the annular ligament of the radial head becoming stretched and entrapped. There is usually a history of a pull on the affected arm, su</p>
<p>ch as when a child tries to run off in a different direction when walking with the hand held by a parent.Sometimes the incident is unobserved or thought to be too trivial to have caused any injury. The child typically allows the arm to hang loosely by their side in a pronated position. They are usually undistressed unless the arm is moved.</p>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/pulled_elbow001.jpg"><img class="size-medium wp-image-285 alignnone" title="pulled_elbow001" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/pulled_elbow001-300x200.jpg" alt="pulled_elbow001" width="243" height="204" /></a></h2>
<h2>Clinical Features</h2>
<ul>
<li>The parents come with complaint of the child not using the affected limb</li>
<li>here is usually a history of a pull on the affected arm, such as when a child tries to run off in a different direction when walking with the hand held by a parent. Although there may not be history of pull in half the cases</li>
<li>Inspection: The child keeps the elbow in extension and the forearm in pronation and is distressed only on elbow movement. Usually there is no swelling, deformity or bruising of the elbow or wrist</li>
<li>Palpation:  tenderness is usually absent</li>
<li>Movements: marked resistance and pain with supination of the forearm.</li>
</ul>
<h3>Diagnosis</h3>
<ul>
<li>Clinically established with a classic history and examination.</li>
<li>X rays are unnecessary if there is a typical history and no visible swelling or deformity. If the child has a pulled elbow the X ray is normal. The child may have normal use of the arm on return from radiology since positioning by the radiographer may solve the problem.</li>
<li>Plain radiographs are indicated when a differential diagnosis is suspected:
<ul>
<li>significant tenderness, swelling, bruising or deformity</li>
<li>reduction fails</li>
</ul>
</li>
</ul>
<h2>Treatment</h2>
<p>The treatment consists of reducing the ligament back to its original position</p>
<ol id="intelliTxt">
<li>Have your child sit in a chair facing you or stand facing you. Ask an older sibling or adult to distract the child as that there is slight temporary pain when you treat a pulled elbow, and it will be easier to slip the ligament back into place if the child is relaxed.<a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/grasping_elbow_closed1.jpg"><img class="alignnone size-full wp-image-291" title="grasping_elbow_closed" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/grasping_elbow_closed1.jpg" alt="grasping_elbow_closed" width="181" height="146" /></a></li>
<li>You should support the elbow with one of your hands, placing your thumb over the radial head with some gentle pressure. Hold<br />
the child’s hand in your other hand as if you are shaking hands.</li>
<li>Fully supinate the forearm and then fully flex the elbow. A click is usually felt over the radial head either when the elbow is fully supinated or fully flexed. This is known as supination/flexion manoeuvre. Alternatively one can also try full pronation followed by flexion (pronation flexion manoeuvre).<br />
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/supination_only.jpg"><img class="size-full wp-image-286 alignnone" title="supination_only" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/supination_only.jpg" alt="supination_only" width="170" height="136" /></a></h2>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/part_flexion1.jpg"><img class="alignnone size-full wp-image-289" title="part_flexion" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/part_flexion1.jpg" alt="part_flexion" width="169" height="135" /></a></h2>
<h2><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/full_flexion.jpg"><img title="full_flexion" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/full_flexion.jpg" alt="full_flexion" width="172" height="137" /></a></h2>
</li>
<li>A click is felt as the ligament moves back into the correct position. You may not always hear this happen.</li>
<li>Offer the child a toy to see if he or she will once again use his or her arm.  A good test for successful reduction is whether the child will actively reach for a toy at arms reach. Most children will use the arm normally within 5 minutes of the reduction. There may be residual pain, but overall, the child should feel much better.</li>
<li>Failure may be due to not putting the elbow through the complete range of motion. Repeat the reduction if the ligament is still out of place. This process should cure and treat your child’s pulled elbow in a few seconds, and they should be ready to play once again.</li>
<li>If the reduction has been delayed for 12 hours or longer the child may not use the arm normally for a longer period of time.  A sling ± backslab can be used for comfort, with review of whether the child will use the arm 24 hours late</li>
</ol>
<h2>Recurrant Pulled Elbow</h2>
<p>Recurrances with pulled elbow are common till 3-4 years age. The repeat injuries are treated in the similar manner. Repeated subluxations do not produce long term problems. The problem generally disappears by 5 year age. Very rarely, in child with multiple recurrances the doctor may advice cast for 3 weeks</p>
]]></content:encoded>
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		<title>FAQs on Ponseti treatment for clubfoot</title>
		<link>http://ipodindia.org/2009/08/faqs-on-ponseti-treatment-for-clubfoot/</link>
		<comments>http://ipodindia.org/2009/08/faqs-on-ponseti-treatment-for-clubfoot/#comments</comments>
		<pubDate>Mon, 31 Aug 2009 01:37:47 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Club foot]]></category>
		<category><![CDATA[Featured]]></category>
		<category><![CDATA[Orthopaedician's Section]]></category>
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		<category><![CDATA[Post graduate Corner]]></category>

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		<description><![CDATA[what is new in the treatment of clubfoot Answered in frequently asked questions mode with answers from recent literature]]></description>
			<content:encoded><![CDATA[<h2><em>Answered by the evidence from the literature</em></h2>
<p><strong>-Taral Nagda</strong></p>
<p><strong>-Rajeev Niravane</strong></p>
<p>Institute of Paediatric Orthopaedic Disorders</p>
<p><a href="http://www.ipodindia.org/">www.ipodindia.org</a></p>
<p>taralnagda@gmail.com</p>
<h3>Who should do Ponseti treatment?</h3>
<p>v      <strong>J Bone Joint Surg Am. 2009 May;91(5):1101-8.<br />
Comparison of surgeon and physiotherapist-directed Ponseti treatment of idiopathic clubfoot.<br />
Janicki JA, Narayanan UG, Harvey BJ, Roy A, Weir S, Wright JG.</strong></p>
<ul>
<li>The introduction of the physiotherapist-supervised clubfoot clinic has been effective without compromising the quality of care of children with clubfoot deformity.</li>
</ul>
<p>v      <strong>Ann R Coll Surg Engl. 2007 Jul;89(5):510-2.Ponseti treatment in the management of clubfoot deformity &#8211; a continuing role for paediatric orthopaedic services in secondary care centres.Docker CE, Lewthwaite S, Kiely NT.</strong></p>
<ul>
<li>Similar results between tertiary Ped ortho dept and physiotherpy dept</li>
</ul>
<p>v      <strong>J Bone Joint Surg Br. 2006 Aug;88(8):1085-9.<br />
Early results of a physiotherapist-delivered Ponseti service for the management of idiopathic congenital talipes equinovarus foot deformity.<br />
Shack N, Eastwood DM.</strong></p>
<ul>
<li>Ponseti technique is suitable for use by non-medical personnel, but a holistic approach and good continuity of care are essential to the success of the programme</li>
</ul>
<h3>Can neglected CTEV be treated with Ponseti method?</h3>
<p>v      <strong>J Pediatr Orthop B. 2009 Mar;18(2):76-8.Results of treatment of idiopathic clubfoot in older infants using the Ponseti method: a preliminary report.</strong></p>
<p><strong>Hegazy M, Nasef NM, Abdel-Ghani H.</strong></p>
<p>The use of thePonseti method in older-aged infants with idiopathic congenital clubfoot seems to</p>
<p>be an effective method of treatment, obviating the need for extensive surgery.</p>
<p>v      <strong>J Bone Joint Surg Br. 2007 Mar;89(3):378-81.<br />
Correction of neglected idiopathic club foot by the Ponseti method.<br />
LourenÃ§o AF, Morcuende JA. Brazil</strong><strong></strong></p>
<p>Only 5/24 needed surgery</p>
<ul>
<li>Ponseti method is a safe, effective and low-cost treatment for neglected idiopathic club foot presenting after walking age.</li>
</ul>
<p>v      <strong>Clin Orthop Relat Res. 2006 Mar;444:224-8.<br />
Ponseti management of clubfoot in older infants.<br />
</strong><strong>Bor N, Herzenberg JE, Frick SL. Israel</strong></p>
<p>older infants with clubfoot can be treated successfullywithout extensive surgery. Our results in older infants are similar to theresults of a previous study we conducted with younger infants</p>
<p>3 % rate of surgery in children less than 3 years</p>
<p>v      <strong>Arch Orthop Trauma Surg. 2006 Jan;126(1):15-21. Epub 2005 Nov 10.<br />
Ponseti technique for the correction of idiopathic clubfeet presenting up to 1 year of age. A preliminary study in children with untreated or complexdeformities.<br />
GÃ¶ksan SB Turkey</strong></p>
<ul>
<li>Our results show that the Ponseti technique is reproducible and effective in children at least up to 12months of age</li>
<li>Only 3 % need extensive PMR</li>
</ul>
<h3>Ponseti method in post PMR cases??</h3>
<p>v      <strong>Clin Orthop Relat Res. 2009 May;467(5):1298-305. Epub 2009 Feb 4.<br />
Is it possible to treat recurrent clubfoot with the Ponseti technique after posteromedial release?: a preliminary study.<br />
Nogueira MP, Ey Batlle AM, Alves CG.</strong></p>
<ul>
<li>initial and final Pirani scores and range of motion of the ankle and subtalar joint. Plantigrade and fully corrected feet were obtained in 71 feet (86%); 11 feet obtained partial correction; one patient failed treatment and underwent another posteromedial release. Recurrences occurred in ninepatients (12 feet or 14%)</li>
</ul>
<h3>Does FAB give rise to femur/ knee/ tibial tortional deformities ?</h3>
<p>v      <strong>J Pediatr Orthop. 2007 Sep;27(6):712-6.<br />
Foot abduction brace in the Ponseti method for idiopathic clubfoot deformity: torsional deformities and compliance.<br />
Boehm S, Sinclair M.</strong></p>
<ul>
<li>Application of the foot abduction brace did not result in pathological changes of femoral anteversion or tibial torsion</li>
</ul>
<h3>Can Ponseti method avoid surgery?</h3>
<p>v      <strong>Clin Orthop Relat Res. 2009 Apr 7.<br />
Comparison of Ponseti versus Surgical Treatment for Idiopathic Clubfoot: A Short-term Preliminary Report.<br />
Zwick EB, Kraus T, Maizen C, Steinwender G, Linhart WE.</strong></p>
<ul>
<li>a favorable short-term outcome for the Ponseti method when compared with a traditional treatment protocol</li>
</ul>
<p>v      <strong>Clin Orthop Relat Res. 2009 May;467(5):1271-7. Epub 2009 Jan 14.<br />
Ponseti method: does age at the beginning of treatment make a difference?<br />
</strong><strong>Alves C, Escalda C, Fernandes P, Tavares D, Neves MC.</strong></p>
<ul>
<li>according to their age at the  beginning of treatment; Group I was younger than 6 months and Group II was &gt; 6 months.</li>
<li>The rate of the Ponseti method in avoiding extensive surgery was 100% in Groups I and II;</li>
<li>relapses occurred in 8% of the feet in older children</li>
</ul>
<p>v      <strong>J Pediatr Orthop B. 2007 Sep;16(5):317-21.<br />
</strong><strong>Comparative results of the conservative treatment in clubfoot by two different protocols.<br />
</strong><strong>Cosma D, Vasilescu D, Vasilescu D, Valeanu M.</strong></p>
<ul>
<li>The Ponseti method decreases the number of surgical interventions needed for the correction of the deformation compared with our traditional method.  5 % need surgery</li>
</ul>
<p>v      <strong>Z Orthop Ihre Grenzgeb. 2006 Sep-Oct;144(5):497-501.<br />
Treatment of congenital clubfoot with the Ponseti method<br />
Eberhardt O, Schelling K, Parsch K, Wirth T.</strong></p>
<ul>
<li>With the Ponseti methodthe need for extensive corrective surgery is greatly reduced. (2/41)</li>
</ul>
<p>v      <strong>Pediatrics. 2004 Feb;113(2):376-80.<br />
Radical reduction in the rate of extensive corrective surgery for clubfoot using the Ponseti method.<br />
Morcuende JA, Dolan LA, Dietz FR, Ponseti IV</strong></p>
<ul>
<li>11 % relapse</li>
<li>3 % extensive surgery rate</li>
</ul>
<h3>What are the factors affecting relapse?</h3>
<p>v      <strong>J Child Orthop. 2009 Jun 3.<br />
Improved bracing compliance in children with clubfeet using a dynamic orthosis.<br />
Garg S, Porter K.</strong></p>
<ul>
<li>Non-compliance with foot abduction bracing in children with clubfeet treated with the Ponseti method is the leading risk factor for deformity recurrence.</li>
<li>A dynamic foot abduction orthosis is believed to result in improved compliance, fewer skin complications, and fewer recurrences</li>
</ul>
<p>v      <strong>J Bone Joint Surg Am. 2007 Mar;89(3):487-93.<br />
Early clubfoot recurrence after use of the Ponseti method in a New Zealand population.<br />
Haft GF, Walker CG, Crawford HA.</strong></p>
<ul>
<li>Compliance with the postcorrection abduction bracing protocol is crucial to avoid recurrence of a clubfoot deformitytreated with the Ponseti method.</li>
<li> When the parents comply with the bracingprotocol, the Ponseti method is very effective at maintaining a correction,although minor recurrences are still common.</li>
<li>When the parents do not comply with the bracing protocol, many major and minor recurrences should be expected</li>
</ul>
<p>v      <strong>Iowa Orthop J. 2007;27:82<br />
</strong><strong>Neuromuscular disease as the cause of late clubfoot relapses: report of 4 cases.<br />
Lovell ME, Morcuende JA.</strong></p>
<ul>
<li>Late relapses in patients with idiopathic clubfoot may represent the onset of a previously undiagnosed neuromuscular disease, and should be thoroughly evaluated.</li>
</ul>
<p>v      <strong>J Pediatr Orthop. 2005 Mar-Apr;25(2):225-8.Use of the foot abduction orthosis following Ponseti casts: is it essential?<br />
</strong><strong>Thacker MM, Scher DM, Sala DA, van Bosse HJ, Feldman DS, Lehman WB</strong></p>
<ul>
<li>The feet of patients compliant with FAOuse remained better corrected than the feet of those patients who were not compliant</li>
</ul>
<p>v      <strong>J Bone Joint Surg Am. 2004 Jan;86-A(1):22-7.<br />
Factors predictive of outcome after use of the Ponseti method for the treatment of idiopathic clubfeet.<br />
Dobbs MB, Rudzki JR, Purcell DB, Walton T, Porter KR, Gurnett CA.</strong></p>
<ul>
<li>Noncompliance and the educational level of the parents(high-school education or less) are significant risk factors for the recurrenceof clubfoot deformity after correction with the Ponseti method. The identification of patients who are at risk for recurrence may allow intervention to improve the compliance of the parents with regard to the use of orthotics,and, as a result, improve outcome</li>
</ul>
<h3>How does one treat a replapse?</h3>
<p>v      <strong>Instr Course Lect. 2006;55:625-9.<br />
Treatment of a recurrent clubfoot deformity after initial correction with the Ponseti technique.<br />
Dietz FR.Department of Orthopaedics, University of Iowa, Iowa City, Iowa, USA.</strong></p>
<ul>
<li>The indication for anterior tibial tendon transfer is the presence of dynamic supination during gait. After tendon transfer, bracing is no longer required because the eversion force of the transferred tendon maintainsthe correction</li>
</ul>
<h3>Is Ponseti method applicable to clubfoot with MMC?</h3>
<p>v      <strong>J Bone Joint Surg Am. 2009 Jun;91(6):1350-9.<br />
Early results of the Ponseti method for the treatment of clubfoot associated with myelomeningocele.<br />
Gerlach DJ, Gurnett CA, Limpaphayom N, Alaee F, Zhang Z, Porter K, Kirchhofer M,<br />
Smyth MD, Dobbs MB.</strong></p>
<ul>
<li>Initial correction was achieved in twenty-seven clubfeet (96.4)</li>
<li>Relapse of deformity was detected in 68% of the feet in the myelomeningocele group, compared with 26% of the feet in the idiopathic group (p = 0.001).</li>
<li>Relapses were treated successfully without the need for extensive soft-tissue</li>
</ul>
<p>v      <strong>Journal of Pediatric Orthopaedics. 29(4):393-397, June 2009.<br />
Treatment of Neuromuscular and Syndrome-Associated (Nonidiopathic) Clubfeet Using the Ponseti Method<br />
Joseph A. Janicki, MD,* Unni G. Narayanan, MBBS, MSc, FRCSC,Þ Barbara Harvey, BHScPT,</strong></p>
<ul>
<li>Ponseti method is worth applying to nonidiopathic clubfeet in an attempt to avoid surgical release.</li>
<li>Nonidiopathic clubfeet required significantly more casts (6.4 vs 4.8) to achieve initial correction and had a higher recurrence rate (44% vs 13%).</li>
</ul>
<h3>Is it necessary to change protocol in complex clubfoot ?</h3>
<p><strong>Clin Orthop Relat Res. 2006 Oct;451:171-6.<br />
Treatment of the complex idiopathic clubfoot<br />
Ponseti IV</strong></p>
<ul>
<li>Modifying      the treatment protocol for complex clubfeetsuccessfully corrected the      deformity without the need for extensive correctivesurgery</li>
</ul>
]]></content:encoded>
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		<title>IPOD OUTREACH PROGRAMME</title>
		<link>http://ipodindia.org/2009/07/ipod-outreach-programme/</link>
		<comments>http://ipodindia.org/2009/07/ipod-outreach-programme/#comments</comments>
		<pubDate>Sat, 04 Jul 2009 02:32:21 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[IPOD Focus]]></category>
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		<description><![CDATA[IPOD attempts to reach out to children with orthopaedic problems in different parts of India and also in other countries. We are a team of Paediatric Orthopaedic Surgeons and Rehabilitation Specialists and can help NGOs, Government programmes, Private Hospitals, Rehabilitation centres. The programme may be in any form -outreach clinics, assessment camps, surgical camps, lectures, workshop, awareness campaigns etc.  If you want to be a part of IPOD's outreach activities please email to taralnagda@gmail.com. We would like to know your profile and in what ways you want to be associated with IPOD. ]]></description>
			<content:encoded><![CDATA[<h1><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/outreach1.jpg"></a><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/baby_hand_reaching1-full.jpg"><img class="alignnone size-full wp-image-276" title="baby_hand_reaching1-full" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/baby_hand_reaching1-full.jpg" alt="baby_hand_reaching1-full" width="284" height="188" /></a></h1>
<p><span style="color: #888888;">Appointments 09320642121 Helpline 09321329888 Emergency 09320129888</span><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/outreach1.jpg"><br />
</a></p>
<table class="MsoNormalTable" style="margin: 2.75pt 6.75pt; border-collapse: collapse;" border="0" cellspacing="0" cellpadding="0" align="left">
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<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Rajkot</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span lang="SV">Dr D K Shah Hosp</span></p>
<p style="margin: 0in 0in 0.0001pt;"><span lang="SV">Om Babycare Hosp</span></p>
<p style="margin: 0in 0in 0.0001pt;"><span lang="SV">Mangalam Hosp</span></p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">26 September</span></p>
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">2009<br />
</span></td>
</tr>
<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Solapur</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Kothadia Hospital</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">12 September 2009</span></p>
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<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Aurangabad</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Patwardhan Hosp</p>
<p style="margin: 0in 0in 0.0001pt;">Raghavendra Hosp</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">19th September 2009</span></p>
</td>
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<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Bhavnagar</strong></span></h3>
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<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">PNR Society</p>
<p style="margin: 0in 0in 0.0001pt;">Jalian Clinic</p>
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<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">6th,7th November  2009</span></p>
</td>
</tr>
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<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Surat</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Nirmal Hospital</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">4th October 2009</span></p>
</td>
</tr>
<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Jamnagar</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Maheshwari Orthopaedic Hospita</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">25 September 2009</span></p>
</td>
</tr>
<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Indore</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Choitharam Hosp and Research Centre</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;"> 27 28 Nov2009</span></p>
</td>
</tr>
<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Kota</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Akansha Orthopaedic Hospital</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">22<sup>nd</sup> September 2009</span></p>
</td>
</tr>
<tr>
<td style="padding: 0in 5.4pt; width: 94.25pt;" width="126" valign="top">
<h3><span style="color: #993300;"><strong>Bhopal</strong></span></h3>
</td>
<td style="padding: 0in 5.4pt; width: 145.15pt;" width="194" valign="top">
<p style="margin: 0in 0in 0.0001pt;">Chirayu Hospital</p>
</td>
<td style="padding: 0in 5.4pt; width: 99pt;" width="132" valign="top">
<p style="margin: 0in 0in 0.0001pt;"><span style="color: #3366ff;">23<sup>rd</sup> September 2009</span></p>
</td>
</tr>
</tbody>
</table>
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		<title>Closed Reduction and Percutaneous Pinning for Supra Condylar fractures of Humerus in Children</title>
		<link>http://ipodindia.org/2009/07/109/</link>
		<comments>http://ipodindia.org/2009/07/109/#comments</comments>
		<pubDate>Fri, 03 Jul 2009 17:10:14 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Patient's Section]]></category>

		<guid isPermaLink="false">http://ipodindia.org/?p=109</guid>
		<description><![CDATA[
Introduction:
Supracondylar fractures are the third most common fractures in children but at the same time the most commonly operated ones. Closed reduction and percutaneous pinning (CRPP) has become the gold standard of treatment of the supracondylar fractures in children preventing complications of deformities and compartment syndrome.
Indications:
1.	All grade 3: CRPP is the gold standard of treatment. [...]]]></description>
			<content:encoded><![CDATA[<p><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/supracondylar_fracture001.jpg"></a><a href="http://ipodindia.org/wp-content/themes/tma/images/uploads/supracondylar_fracture0011.jpg"><img class="alignnone size-full wp-image-125" title="supracondylar_fracture0011" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/supracondylar_fracture0011.jpg" alt="supracondylar_fracture0011" width="141" height="163" /></a></p>
<h2><span style="color: #ff00ff;">Introduction:</span></h2>
<p>Supracondylar fractures are the third most common fractures in children but at the same time the most commonly operated ones. Closed reduction and percutaneous pinning (CRPP) has become the gold standard of treatment of the supracondylar fractures in children preventing complications of deformities and compartment syndrome.<br />
Indications:<br />
1.	All grade 3: CRPP is the gold standard of treatment. Even in the patients with vascular injury, open fractures and nerve injury it is the initial line of treatment.<br />
2.	Grade 2 when reduced because reduction process breaks the posterior cortex and converts type 2 into unstable type 3.<br />
3.	Grade 1 with medial comminution as they have tendenct to produce varus deformity if not fixed.</p>
<h2><span style="color: #ff00ff;"><strong>Princinciples:</strong></span></h2>
<p>The most common complication of SCSH is cubitus varus, and the most dreaded complication is Volkmann ischemic contracture. The first can be prevented by an accurate reduction. However, without fixation, it is necessary to maintain the elbow at 110 degrees or more of flexion to maintain the reduction. With the subsequent swelling that occurs around the elbow, this position restricts venous drainage from the forearm, resulting in increased swelling and compartment syndrome. If the elbow is extended to 90 degrees, there is a likelihood that the reduction will be lost. An accurate reduction and the use of percutaneous skeletal fixation to permit the elbow to extend to 90 degrees while maintaining reduction thus prevents both the complications allowing optimum treatment of these fractures in children.</p>
<h2><strong><span style="color: #ff00ff;">Biomechanics Of Reduction </span></strong></h2>
<p>To understand the importance of accurate reduction, it is necessary to understand peculiar of the humerus at the fracture site and the mechanics of how the deformity occurs.<br />
The supracondylar fracture of the humerus occurs through the thinnest portion of the bone: the area of the coronoid fossa anteriorly and the coracoid fossa posteriorly. A cross-section of this area looks like a dum bell like structure with thick medial and lateral bone masses and thin central area in between. Therefore, without the rotational correction, the medial and lateral columns will not be opposed and naturally and the  reduction will be unstable. As a result, the distal fragment may tilt and angulate, producing deformity. This rotational deformity can be seen clinically in cases of angular deformity. However, because of the large range of rotation in the shoulder joint, this rotational deformity is not apparent cosmetically or functionally.</p>
<h2><strong><span style="color: #ff00ff;">OT Arrangement</span></strong></h2>
<p>It must be emphasized that this is a radiographic technique. Proper positioning of the patient, the image intensifier, and the assistant greatly simplifies the procedure.</p>
<p>The recording tube of the image intensifier becomes the operating table. The patient is shifted to the side of the table so that the shoulder is at the edge. A tourniquet can be applied in case an open reduction is necessary. The arm is draped. A sheet is kept under the arm to fascilitate the use of drill for K wire fixation.<strong><br />
</strong></p>
<h2><span style="color: #ff00ff;"><strong>The Technique of Reduction</strong></span></h2>
<p>It is important to remember that this reduction does not require strength, and in fact, if the reduction is not done gently, the remaining periosteal hinge will be torn, creating an unstable situation and a much more difficult reduction.<br />
The reduction of a supracondylar fracture is not difficult if each of the steps in the reduction described by Rang are performed in the proper sequence.<br />
<strong>Step 1 Traction: </strong>The first step in the reduction is the application of traction to the arm, which is flexed about 15 degrees to reduce the distal fragment in line with the proximal fragment. In most extension type supracondylar fractures, there is an intact periosteum posteriorly that aids in the reduction by providing a stable fulcrum.</p>
<p><strong>Step 2 Correction of medio-lateral displacement:</strong> The second step is the correction of the medial or lateral displacement. This can be seen from the preoperative radiograph or the image intensifier during the reduction. It must be performed before the surface of the two bony fragments are brought into contact because after they are reduced, it is impossible to slide one over the other.<strong><br />
Step 3 Correction of Rotataion:</strong> The third step is to correct the rotation. This must be done again before the two fragments are brought into apposition. If the rotation is not correct, the fragments must be disengaged before another attempt at correction of the rotation. In most cases, the distal fragment requires external rotation.<br />
<strong>Step 4 Correction of posterior displacement and locking of reduction: </strong>With rotation and medial and lateral displacement corrected, the posterior displacement can be corrected. While the assistant supports the arm, the surgeon places one thumb behind the medial condyle and the other behind the lateral condyle. Pushing forward, the surgeon corrects the posterior displacement. The elbow is then flexed, which reduces the fracture. The final step of reduction is to pronate (if medially displaced) or supinate (if laterally displaced) the arm to tighten the remaining medial or lateral periosteal hinge.</p>
<h2><span style="color: #ff00ff;">THE TECHNIQUE OF PINNING</span></h2>
<p>The assistant holds the elbow in acute flexion, which maintains the reduction.<br />
The arm is examined with the image intensifier to determine the adequacy of the reduction. Four views should be examined by internally and externally rotating the arm back and forth at each view to ensure that the proper aspect of the reduction is seen. The anteroposterior view is the least helpful because the overlying forearm obscures the bony detail of the humerus. The arm is held acutely flexed while the lateral view is examined. If the diameters of the two fragments at the fracture site are different, malrotation is present. If the proper angle between the condyles and the shaft has not been restored, hyperextension and lack of full flexion of the elbow results. Next, the lateral and medial condyles are examined by oblique views. The internal oblique view demonstrates the lateral condyle, and the external oblique view demonstrates the medial condyle. If the lateral view shows that the rotation is incompletely reduced, these oblique views demonstrate which condyle is not completely reduced and whether more internal or external rotation is needed.</p>
<p>When the reduction is achieved, the percutaneous pins are inserted. Usually, 1.25 Kirschner wires are adequate for the fixation.</p>
<p>The options for pin placement are<br />
1.	Two lateral pins<br />
2.	Three lateral pins<br />
3.	One medial and one lateral pin<br />
One medial and one lateral pin provide the optimal degree of stability but carries a small risk of ulnar nerve injury. We pass two or three lateral pins and do external rotation test to stability of ulnar nerve. Following this is the fixation is stable medial pin is not passed. However if there is medial comminution or if the fixation is unstable additional medial pin is passed taking care not to injure the ulnar nerve</p>
<p><strong>Lateral K wire Insertion<br />
</strong>Although it is more easier to rotate the arm externally than internally and pass medial pin forst the position of hyperflexion subluxates the ulnar nerve anteriorly making it vulnerable to injury. Hence the lateral pin is passed first.<br />
The assistant continues to hold the elbow acutely flexed while rotating the arm with the lateral condyle facing up. The lateral condyle is palpated, and the Kirschner wire on the drill is pushed through the skin and into the bone. The image intensifier can be used to determine whether the location is correct and to aid in directing the wire so that it does not pass too far anteriorly or posteriorly and miss the proximal fragment. The wire should engage the opposite cortex. The direction of wire is from anterior to posterior around 15 degrees to match the lower end humerus profile.</p>
<p><strong>Medial K wire insertion<br />
</strong>The arm, still held in acute flexion, is rotated until the medial condyle faces up. The thumb of the left hand (right-handed surgeon) finds the medial epicondyle. If swelling makes this difficult, the elbow can be extended slightly. The thumb should roll off of the inferior edge of the epicondyle into the ulnar grove and remain there holding the nerve in the grove. The Kirschner wire is started just at the tip of the thumb and is directed with image intensifier guidance, as in the previous example. If the surgeon is uncertain about the location of the ulnar nerve, a small incision over the ulnar groove can be made and the ulnar nerve located and held out of the way with a small curved hemostat.<br />
Both pins should engage the opposite cortex. Ideally, they pass up the medial and lateral columns of bone and do not cross at the fracture site. It is important to ascertain that the pins are in the distal fragment because, in the surgeon&#8217;s desire to avoid the ulnar nerve, the pin may start too far proximally and miss the distal fragment. These views from the image intensifier are recorded as the final films because they will be far superior to any films taken through the cast, and no further change in the fracture can occur if the pins are placed properly. Finally, the arm can be extended fully and examined clinically for any deformity.<br />
Four View Xray Examination<br />
After placement of both pins, the arm is again examined in all four views with the image intensifier to confirm the reduction and the placement of the pins. The internal rotation oblique view demonstrates the lateral condyle, and the external oblique view  demonstrates the medial condyle. If the fracture is not anatomically reduced, these views show which condyle is incompletely reduced.<br />
If all is satisfactory, the pins are cut and bent over, leaving them outside the skin to facilitate removal in the office.</p>
<h2><span style="color: #ff00ff;">Post op management</span></h2>
<p>The arm is held in 90 degrees of elbow flexion and supination. Above elbow slab is given in 100 degrees of flexion. Pins are removed at 4 weeks after confirming union and elbow mobilization is started</p>
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		<title>Osteogenesis Imperfecta Types</title>
		<link>http://ipodindia.org/2009/07/osteogenesis-imperfecta-types/</link>
		<comments>http://ipodindia.org/2009/07/osteogenesis-imperfecta-types/#comments</comments>
		<pubDate>Thu, 02 Jul 2009 02:55:50 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Featured]]></category>
		<category><![CDATA[Orthopaedician's Section]]></category>
		<category><![CDATA[Osteogenesis Imperfecta]]></category>
		<category><![CDATA[Patient's Section]]></category>
		<category><![CDATA[Post graduate Corner]]></category>

		<guid isPermaLink="false">http://ipodindia.org/?p=67</guid>
		<description><![CDATA[Since 1979, OI has been classified by type according to a scheme developed by David Sillence,
M.D. This system is based on mode of inheritance, clinical picture, and radiologic appearance.
The OI type descriptions provide some information to the clinician and family about a person’s
prognosis, but they do not predict functional outcome. It is also important to note that the
severity of OI ranges greatly; the different types of OI represent somewhat arbitrary cutoffs
along a continuum. As a result, the severity of the disorder may vary significantly among people
who have the same type. The OI classification scheme has continued to evolve as new
information about OI is discovered.
]]></description>
			<content:encoded><![CDATA[<p><img class="alignnone size-medium wp-image-80" title="TYPES OF OI" src="http://ipodindia.org/wp-content/themes/tma/images/uploads/12f1-300x167.jpg" alt="TYPES OF OI" width="300" height="167" /></p>
<h2><span style="color: #ff00ff;"><strong>Type I:</strong></span></h2>
<p><strong>• OI Type I is the mildest and most common form of the disorder. It accounts for 50<br />
percent of the total OI population.<br />
• Type I manifests with mild bone fragility, relatively few fractures, and minimal limb<br />
deformities. The child might not fracture until he or she is ambulatory.<br />
• Shoulders and elbow dislocations may occur more frequently in children with OI than in<br />
healthy children.<br />
• Some children have few obvious signs of OI or fractures. Others experience multiple<br />
fractures of the long bones, compression fractures of the vertebrae, and chronic pain.<br />
• The intervals between fractures may vary considerably.<br />
• After growth is completed, the incidence of fractures decreases considerably.<br />
• Blue sclerae are often present.<br />
Guide to Osteogenesis Imperfecta for Pediatricians and Family Practice Physicians 6<br />
• Typically, a child’s stature may be average or slightly shorter than average as compared<br />
with unaffected family members, but is still within the normal range for the age.<br />
• There is a high incidence of hearing loss. Onset occurs primarily in young adulthood, but<br />
it may occur in early childhood.<br />
• Dentinogenesis imperfecta is often absent.<br />
• OI Type I is dominantly inherited. It can be inherited from an affected parent, or, in<br />
previously unaffected families, it results from a spontaneous mutation. Spontaneous<br />
mutations are common.<br />
• Biochemical tests on cultured skin fibroblasts show a lower-than-normal amount of type I<br />
collagen. Collagen structure is normal.<br />
• People with OI Type I experience the psychological burden of appearing normal and<br />
healthy to the casual observer despite needing to accommodate their bone fragility.<br />
• The absence of obvious symptoms in some children may contribute to problems at school<br />
or with peers.<br />
• Significant care issues that arise with OI Type I include gross motor developmental<br />
delays, joint and ligament weakness and instability, muscle weakness, the need to prevent<br />
fracture cycles, and the necessity of spine protection. (See “Behavioral and Lifestyle<br />
Modifications,” page 13.) Children with OI and their parents will need emotional support<br />
at each new developmental stage. Family members should carry documentation of the OI<br />
diagnosis to avoid accusations of child abuse at emergency rooms.<br />
• The treatment plan should maximize mobility and function, increase peak bone mass, and<br />
develop muscle strength. Physical therapy, early intervention programs, and as much<br />
exercise and physical activity as possible will improve outcomes.<br />
</strong></p>
<h2><span style="color: #ff00ff;"><strong>Type II:</strong></span></h2>
<p><strong>• OI Type II is the most severe form.<br />
• At birth, infants with OI Type II have very short limbs, small chests, and soft skulls.<br />
Their legs are often in a frog-leg position.<br />
• The radiologic features are characteristic and include absent or limited calvarial<br />
mineralization; flat vertebral bodies; very short, telescoped, broad femurs; beaded and<br />
often broad short ribs; and evidence of malformation of the long bones.<br />
• Intrauterine fractures will be evident in the skull, long bones, or vertebrae.<br />
• The sclerae are usually very dark blue or gray.<br />
• The lungs are underdeveloped.<br />
• Infants with OI Type II have low birth weights.<br />
• Respiratory and swallowing problems are common.<br />
• Macrocephaly may be present. Microcephaly is rarely present.<br />
• Infants with OI Type II usually die within weeks of delivery. A few may survive longer;<br />
they usually die of respiratory and cardiac complications.<br />
• OI Type II results from a new dominant mutation in a type 1 collagen gene or parental<br />
mosaicism. Similar extremely severe types of OI, Types VII and VIII, can be caused by<br />
recessive mutations to other genes. (See “Type VII” and “Type VIII,” pages 9 and 10.)<br />
• Genetic counseling is recommended for parents of a child with OI Type II before any<br />
future pregnancies.<br />
• Significant care issues that arise with OI Type II include obtaining an accurate diagnosis,<br />
Guide to Osteogenesis Imperfecta for Pediatricians and Family Practice Physicians 7<br />
getting genetic counseling, the family’s need for emotional support, and management of<br />
respiratory and cardiac impairments. Infants with OI Type II who can breathe without a<br />
respirator and those with severe OI Type III may be candidates for off-label treatment<br />
with bisphosphonates. At this time, pamidronate (Aredia*) is the only bisphosphonate<br />
that has been studied in infants who have OI. Treatment research is ongoing. (See “Drug<br />
Therapies – Bisphosphonates,” page 15.)<br />
*Brand names included in this publication are provided as examples only, and their inclusion does not mean<br />
that these products are endorsed by the National Institutes of Health or any other Government agency. Also, if a<br />
particular brand name is not mentioned, this does not mean or imply that the product is unsatisfactory.<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type III:</span></strong></h2>
<p><strong>• OI Type III is the most severe type among children who survive the neonatal period. The<br />
degree of bone fragility and the fracture rate vary widely.<br />
• This type is characterized by structurally defective type I collagen. This poor-quality type<br />
I collagen is present in reduced amounts in the bone matrix.<br />
• At birth, infants generally have mildly shortened and bowed limbs, small chests, and a<br />
soft calvarium.<br />
• Respiratory and swallowing problems are common in newborns.<br />
• There may be multiple long-bone fractures at birth, including many rib fractures.<br />
• Frequent fractures of the long bones, the tension of muscle on soft bone, and the<br />
disruption of the growth plates lead to bowing and progressive malformation. Children<br />
have a markedly short stature, and adults are usually shorter than 3 feet, 6 inches, or<br />
102 centimeters.<br />
• Spine curvatures, compression fractures of the vertebrae, scoliosis, and chest deformities<br />
occur frequently.<br />
• The altered structure of the growth plates gives a popcorn-like appearance to the<br />
metaphyses and epiphyses.<br />
• The head is often large relative to body size.<br />
• A triangular facial shape, due to overdevelopment of the head and underdevelopment of<br />
the face bones, is characteristic.<br />
• The sclerae may be white or tinted blue, purple, or gray.<br />
• Dentinogenesis imperfecta is common but not universal.<br />
• The majority of OI Type III cases result from dominant mutations in type I collagen<br />
genes. Often these mutations are spontaneous. Similar extremely severe types of OI,<br />
Types VII and VIII, are caused by recessive mutations to other genes. (See “Type VII”<br />
and “Type VIII,” pages 9 and 10.)<br />
• Genetic counseling is recommended for asymptomatic parents of a child with OI Type III<br />
before any future pregnancies.<br />
• Significant care issues that arise with OI Type III include the need to prevent fracture<br />
cycles; the appropriate timing of rodding surgery; scoliosis monitoring; respiratory<br />
function monitoring; the need to develop strategies to cope with short stature and fatigue;<br />
the family’s need for emotional support, especially during the patient’s infancy; and the<br />
off-label use of bisphosphonates. (See “Drug Therapies – Bisphosphonates,” page 15.)<br />
• It is also important to address difficulties with social integration, participation in leisure<br />
activities, and maintaining stamina.<br />
• The treatment plan should maximize mobility and function, increase peak bone mass and<br />
Guide to Osteogenesis Imperfecta for Pediatricians and Family Practice Physicians 8<br />
muscle strength, and employ as much exercise and physical activity as possible.<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type IV:</span></strong></h2>
<p><strong>• People with OI Type IV are moderately affected. Type IV can range in severity from<br />
relatively few fractures, as in OI Type I, to a more severe form resembling OI Type III.<br />
• The diagnosis can be made at birth but often occurs later.<br />
• The child might not fracture until he or she is ambulatory.<br />
• People with OI Type IV have moderate-to-severe growth retardation, which is one factor<br />
that distinguishes them clinically from people with Type I.<br />
• Bowing of the long bones is common, but to a lesser extent than in Type III.<br />
• The sclerae are often light blue in infancy, but the color intensity varies. The sclerae may<br />
lighten to white later in childhood or early adulthood.<br />
• The child’s height may be less than average for his or her age.<br />
• Short humeri and femora are common.<br />
• Long bone fractures, vertebral compression, scoliosis, and ligament laxity may also<br />
be present.<br />
• Dentinogenesis imperfecta may be present or absent.<br />
• OI Type IV has an autosomal dominant pattern of inheritance. Many cases are the result<br />
of a new mutation.<br />
• This type is characterized by structurally defective type I collagen. This poor-quality type<br />
I collagen is present in reduced amounts in the bone matrix.<br />
• Significant care issues that arise with OI Type IV include the need to prevent fracture<br />
cycles; the appropriate timing of rodding surgery; scoliosis monitoring; the need to<br />
develop strategies for coping with short stature and fatigue; the family’s need for<br />
emotional support, especially during the patient’s infancy; and the off-label use of<br />
bisphosphonates. (See “Drug Therapies – Bisphosphonates,” page 15.)<br />
• Family members should carry documentation of the OI diagnosis to avoid accusations of<br />
child abuse at emergency rooms.<br />
• It is also important to address difficulties with social integration, participation in leisure<br />
activities, and maintaining stamina.<br />
• The treatment plan should maximize mobility and function, increase peak bone mass and<br />
muscle strength, and employ as much exercise and physical activity as possible.<br />
Microscopic studies of OI bone, led by Francis Glorieux, M.D., Ph.D., at the Shriners Hospital<br />
for Children in Montreal, have identified a subset of people who are clinically within the OI<br />
Type IV group but have distinctive patterns to their bone. Review of the clinical histories of<br />
these people uncovered other common features. As a result of this research, two types – Type V<br />
and Type VI – were added to the Sillence Classification. Regarding these types, it is important to<br />
note the following:<br />
• They do not involve deficits of type 1 collagen.<br />
• Treatment issues are similar to OI Type IV.<br />
• Diagnosis requires specific radiographic and bone studies.<br />
Guide to Osteogenesis Imperfecta for Pediatricians and Family Practice Physicians 9<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type V:</span></strong></h2>
<p><strong>• OI Type V is moderate in severity. It is similar to OI Type IV in terms of frequency of<br />
fractures and the degree of skeletal deformity.<br />
• The most conspicuous feature of this type is large, hypertrophic calluses in the largest<br />
bones at fracture or surgical procedure sites.<br />
• Hypertrophic calluses can also arise spontaneously.<br />
• Calcification of the interosseous membrane between the radius and ulna restricts forearm<br />
rotation and may cause dislocation of the radial head.<br />
• Women with OI Type V anticipating pregnancy should be screened for hypertrophic<br />
callus in the iliac bone.<br />
• OI Type V is dominantly inherited and represents 5 percent of moderate-to-severe<br />
OI cases.<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type VI:</span></strong></h2>
<p><strong>• OI Type VI is extremely rare. It is moderate in severity and similar in appearance and<br />
symptoms to OI Type IV.<br />
• This type is distinguished by a characteristic mineralization defect seen in biopsied bone.<br />
• The mode of inheritance is probably recessive, but it has not yet been identified.<br />
Recessively Inherited Types of OI (Types VII and VIII):<br />
• Two recessive types of OI, Types VII and VIII, were recently identified. Unlike the<br />
dominantly inherited types, the recessive types of OI do not involve mutations in the type 1<br />
collagen genes.<br />
• These recessive types of OI result from mutations in two genes that affect collagen<br />
posttranslational modification:<br />
― the cartilage-associated protein gene (CRTAP)<br />
― the prolyl 3-hydroxylase 1 gene (LEPRE1).<br />
• Recessively inherited OI has been discovered in people with lethal, severe, and moderate OI.<br />
There is no evidence of a recessive form of mild OI. Recessive inheritance probably accounts<br />
for fewer than 10 percent of OI cases.<br />
• Parents of a child who has a recessive type of OI have a 25 percent chance per pregnancy of<br />
having another child with OI. Unaffected siblings of a person with a recessive type have a 2<br />
in 3 chance of being a carrier of the recessive gene.<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type VII:</span></strong></h2>
<p><strong>• Some cases of OI Type VII resemble OI Type IV in many aspects of appearance and<br />
symptoms.<br />
• Other cases resemble OI Type II, except that infants have white sclerae, small heads and<br />
round faces.<br />
• Short humeri and femora are common.<br />
• Short stature is common.<br />
• Coxa vara is common.<br />
• OI Type VII results from recessive inheritance of a mutation in the CRTAP gene. Partial<br />
(10 percent) expression of CRTAP leads to moderate bone dysplasia. Total absence of the<br />
cartilage-associated protein has been lethal in all identified cases.<br />
Guide to Osteogenesis Imperfecta for Pediatricians and Family Practice Physicians 10<br />
</strong></p>
<h2><strong><span style="color: #ff00ff;">Type VIII:</span></strong></h2>
<p><strong>• Cases of OI Type VIII are similar to OI Types II or III in appearance and symptoms<br />
except for white sclerae.<br />
• OI Type VIII is characterized by severe growth deficiency and extreme undermineralization<br />
of the skeleton.<br />
• It is caused by absence or severe deficiency of prolyl 3-hydroxylase activity due to<br />
mutations in the LEPRE1 gene.<br />
Additional Forms of OI<br />
The following conditions are rare, but they feature fragile bones plus other significant symptoms.<br />
More detailed information on them can be found in Pediatric Bone: Biology and Diseases,<br />
Glorieux et al, 2003.<br />
• Osteoporosis-Pseudoglioma Syndrome: This syndrome is a severe form of OI that also<br />
causes blindness. It results from mutations in the low-density lipoprotein receptor-related<br />
protein 5 (LRP5) gene.<br />
• Cole-Carpenter Syndrome: This syndrome is described as OI with craniosynostosis and<br />
ocular proptosis.<br />
• Bruck Syndrome: This syndrome is described as OI with congenital joint contractures.<br />
It results from mutations in the procollagen-lysine, 2-oxoglutarate 5-dioxygenase 2<br />
(PLOD2) gene encoding a bone-specific lysyl-hydroxylase. This affects collagen<br />
crosslinking.<br />
• OI/Ehlers-Danlos Syndrome: This recently identified syndrome features fragile bones<br />
and extreme ligament laxity. Young children affected by this syndrome may experience<br />
rapidly worsening spine curves.</strong><a href="http://www.shriners.com/Hospitals"><br />
</a></p>
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